Publicaciones académicas
- Neuromotor decline is associated with gut dysbiosis following surgical decompression for Degenerative Cervical Myelopathy. Neurobiology of Disease. https://doi.org/10.1016/j.nbd.2024.106640.
- Regulatory T cells administration reduces anxiety-like behavior in mice submitted to chronic restraint stress. Frontiers in Cellular Neuroscience. https://doi.org/10.3389/fncel.2024.1406832.
- Identification of master regulator genes controlling pathogenic CD4+ T cell fate in inflammatory bowel disease through transcriptional network analysis. Scientific Reports. https://doi.org/10.1038/s41598-024-61158-4.
- Impaired communication at the neuromotor axis during Degenerative Cervical Myelopathy. Frontiers in Cellular Neuroscience. https://doi.org/10.3389/fncel.2023.1316432.
- Degenerative Cervical Myelopathy induces sex-specific dysbiosis in mice. Frontiers in Microbiology. https://doi.org/10.3389/fmicb.2023.1229783.
- Macrophage-based delivery of interleukin-13 improves functional and histopathological outcomes following spinal cord injury. Journal of Neuroinflammation. https://doi.org/10.1186/s12974-022-02458-2.
- Acute Systemic White Blood Cell Changes following Degenerative Cervical Myelopathy (DCM) in a Mouse Model. International Journal of Molecular Sciences. https://doi.org/10.3390/ijms231911496.
- Gut Microbiota–Brain Axis as a Potential Modulator of Psychological Stress after Spinal Cord Injury. Biomedicines. https://doi.org/10.3390/biomedicines10040847.
- IgM Immunoglobulin Influences Recovery after Cervical Spinal Cord Injury by Modulating the IgG Autoantibody Response. eneuro. https://doi.org/10.1523/eneuro.0491-19.2021.
- Gut Microbiota Interaction with the Central Nervous System throughout Life. Journal of Clinical Medicine. https://doi.org/10.3390/jcm10061299.
- Mir21 modulates inflammation and sensorimotor deficits in cervical myelopathy: data from humans and animal models. Brain Communications. https://doi.org/10.1093/braincomms/fcaa234.
- The Cross-Talk Between the Dopaminergic and the Immune System Involved in Schizophrenia. Frontiers in Pharmacology. https://doi.org/10.3389/fphar.2020.00394.
- Targeting the Dopaminergic System in Autoimmunity. Journal of Neuroimmune Pharmacology. https://doi.org/10.1007/s11481-019-09834-5.
- The changes in systemic monocytes in humans undergoing surgical decompression for degenerative cervical myelopathy may influence clinical neurological recovery. Journal of Neuroimmunology. https://doi.org/10.1016/j.jneuroim.2019.577024.
- ADAM17-deficiency on microglia but not on macrophages promotes phagocytosis and functional recovery after spinal cord injury. Brain, Behavior, and Immunity. https://doi.org/10.1016/j.bbi.2019.02.032.
- Endogenous Interleukin-10 Deficiency Exacerbates Vascular Pathology in Traumatic Cervical Spinal Cord Injury. Journal of Neurotrauma. https://doi.org/10.1089/neu.2018.6081.
- Methylprednisolone treatment enhances early recovery following surgical decompression for degenerative cervical myelopathy without compromise to the systemic immune system. Journal of Neuroinflammation. https://doi.org/10.1186/s12974-018-1257-7.
- Delayed decompression exacerbates ischemia-reperfusion injury in cervical compressive myelopathy. JCI Insight. https://doi.org/10.1172/jci.insight.92512.
- Mast cells promote scar remodeling and functional recovery after spinal cord injury via mouse mast cell protease 6. The FASEB Journal. https://doi.org/10.1096/fj.201500114R.
- The Complement Receptor C5aR Has a Dual, Time-Dependent Effect on the Outcome of Spinal Cord Injury. Journal of Neuroscience. https://doi.org/10.1523/JNEUROSCI.2380-15.2015.
- Glycine receptors control the generation of projection neurons in the developing cerebral cortex. Cell Death & Differentiation. https://doi.org/10.1038/cdd.2014.75.
- Immunopharmacological intervention for successful neural stem cell therapy: New perspectives in CNS neurogenesis and repair. Pharmacology & Therapeutics. https://doi.org/10.1016/j.pharmthera.2013.08.001.
- Mast cells protect from post-traumatic spinal cord damage in mice by degrading inflammation-associated cytokines via mouse mast cell protease 4. Neurobiology of Disease. https://doi.org/10.1016/j.nbd.2013.09.012.
- Oncostatin M Reduces Lesion Size and Promotes Functional Recovery and Neurite Outgrowth After Spinal Cord Injury. Molecular Neurobiology. https://doi.org/10.1007/s12035-014-8795-5.
- ADAM17 is a survival factor for microglial cells in vitro and in vivo after spinal cord injury in mice. Cell Death & Disease. https://doi.org/10.1038/cddis.2013.466.
- Glycine Receptor α2 Subunit Activation Promotes Cortical Interneuron Migration. Cell Reports. https://doi.org/10.1016/j.celrep.2013.07.016.
- Late blocking of peripheral TNF-α is ineffective after spinal cord injury in mice. Immunobiology. https://doi.org/10.1016/j.imbio.2012.05.007.
- The role of “anti-inflammatory” cytokines in axon regeneration. Cytokine & Growth Factor Reviews. https://doi.org/10.1016/j.cytogfr.2012.08.008.